Epicritical sensitivity

The epicritical sensitivity is and is a perception system of the skin Sharpness of touch or Fine perception called. It is closely related to proprioception. Disorders of epicritical sensitivity are often caused by peripheral or central nerve damage.

What is the Epicritical Sensitivity?

The human skin senses have different perceptual qualities, which are summarized as surface sensitivity. One of them is epicritical sensitivity. These are the discriminatory perceptions of vibrations, pressure and touch, which are also known as fine perception. In addition, the epicritical sensitivity includes perceptions of the proprioceptive sense of position and is therefore involved both in the interoception of internal stimuli and in the exteroception of external stimuli.

The epicritical sensitivity works with different sensory cells that translate a stimulus into the language of the central nervous system. The epicritic receptors are either exteroceptors or interoceptors. The exteroceptors of epicritical sensitivity are predominantly mechanoreceptors for obtaining information about the localization or fine-tuning of a touch. Proprioceptors such as muscle spindles and tendon spindles are relevant as epicritical interoceptors, which are used to obtain information about the position of muscles and joints.

Protopathic sensitivity must be distinguished from epicritical sensitivity. This second quality of perception of the skin sense provides information on temperatures and pain via thermoreceptors and nociceptors and is also referred to as a predominantly exteroceptive gross perception.

As part of tactile perception, epicritical sensitivity, in contrast to protopathic sensitivity, means the ability to perceive spatially closely spaced touch stimuli as individual stimuli. Fine perception plays a role for both tactile and haptic perception, in the sense of passive and active tactile perception.

Function & task

The epicritical perceptual system is also called the discriminating system of the skin sense. In contrast, the protopathic system of the skin's senses corresponds to a protective system. Epicritical perception can be broken down into passive touch perception and active exploration perception.

All proprioceptive structures in the system are passive structures of perception of touch. The first place for the perception of epicritical information are the receptors. In this context, mechanoceptors such as pressoreceptors and baroreceptors are differentiated from proprioceptors such as muscle spindles. The mechanoceptors are primarily concerned with pressure perception. Proprioceptors are responsible for self-awareness. Beroreceptors, for example, are located in the wall of blood vessels and are involved in the enteroceptive regulation of blood pressure.

Mechanoreceptors are mainly divided into SA, RA and PC receptors. The most important SA receptors are the Merkel cells, the Ruffini corpuscles and the Pinkus Iggo tactile discs for pressure perception. Important RA receptors are the Meissner bodies, the hair follicle sensors and the Krause end bulbs for perception of touch. The Vater-Pacini corpuscles and the Golgi-Mazzoni corpuscles are mainly known as PC receptors for the perception of vibration.

In connection with proprioception, enteroceptive receptors are differentiated from purely proprioceptive receptors. Via enteroceptive epicritical receptors in the bladder, the gastrointestinal tract or the cardiovascular system, automatically controlled body processes such as the urge to urinate, the urge to defecate, the cough reflex or the filling of the auricles are regulated.

All epicritical information is forwarded for all exteroceptive stimuli via the posterior cord tracts of the spinal cord. The protopathic receptors of the skin sense, on the other hand, transmit their information to the cerebellum via the anterior spinocerebellar or posterior posterior spinocerebellar tract. The Hinterstrangbahnen as afferent information path of the epicritical sensibility run uncrossed.

The gracilis fasciculus is responsible for the information that affects the lower extremities. The cuneatus fasciculus, on the other hand, conducts the epicritical information of the upper extremities. The first neuron undergoes a switchover to the second neuron in the nucleus gracilis or nucleus cuneatus of the brain stem. After this switchover, the orbits continue as the lemniscus medialis and cross within the decusatio lemniscorum. In the thalamus they are switched to a third neuron, which then transports the epicritical information to the postcentral gyrus.

As part of tactile perception, epicritical sensitivity in terms of tactile acuity is determined using a two-point discrimination threshold. In young people, the tactile acuity of fine perception is around 1.5 millimeters at the fingertip. In older people it is sometimes only four millimeters. On the back, the tactile acuity of the fine perception is physiologically lowest and amounts to a few centimeters.

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Illnesses & ailments

The most important task of the epicritical system is the assessment and differentiation of key impressions and touch impressions. Disturbances of the epicritical system manifest themselves mainly in the inability to distinguish between touch and touch.

All disorders of the surface sensitivity are most often due to damage to peripheral or central nerves. A lack of sensory integration can also be decisive for disorders of epicritical sensitivity. On the one hand, a sensory integration disorder is due to predisposition and manifests itself in the inability to combine different sensory impressions. On the other hand, it can arise from a lack of physical practice in childhood.

The ability to combine different sensory impressions is particularly crucial for near senses such as the epicritical system and can be increased if necessary. Epicritical sensory disturbances are expressed either as hyperesthesia or as anesthesia. Hyperesthesia corresponds to increased perception or hypersensitivity to touch stimuli and can be painful.

Hyperesthesia often occurs due to acute or chronic irritation of nerve structures, for example after operations or other interventions. Often those affected show a tactile defense that manifests itself in avoiding contact.

The opposite phenomenon is anesthesia, which amounts to numbness. Anesthesia with a local limitation show up, for example, with peripheral polypathies in a certain part of the body, such as those caused by poisoning, diabetes or certain infections. Just as often, local anesthesia can be traced back to damage to the central nervous system as part of a neurological disease such as multiple sclerosis, a stroke or a spinal cord infarction. Traumatic damage to the central nervous system can also be a possible cause. The same applies to tumor diseases of the central nervous system.